Clinical Electrophysiologist

Joined February 2018
13 Photos and videos
Help! Patient for both AF and PVC ablation. Mechanical AVR. On warfarin INR day of procedure 3.2, PTT normal. No baseline ACT. Weight adjusted IV heparin. First ACT over 400. Stayed about 400 for hours despite stopping IV hep. Changed ACT machines too. ??? Also ? Continue hep?
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75 yo, recurrent syncope, rhythm recorded by EMS, baseline ECG at R showing LBBB with first degree. Normal echo, Cath, MRI pending. Thoughts??
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I think other differential includes ARVC missed by echo, atriofascicular pathway, and moderator band VT
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Great thought, given incomplete LBBB in NSR definite possibility and many cases of BB reentry with normal EF described. I do think that the QRS morphology a bit unusual for BB reentry VT and expect more of an R-wave in V6, but EMS ECG
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Do we believe that we can see slow pathway conduction in NSR? Successful ablation of slow pathway at blue markers, His yellow, mapped with Grid
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Can people help me? How do you know EXACTLY where to ablate concealed nodoventricular and fasciculoventricular pathways? VentricularSummit seems too broad.
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I know that in Bundle Branch Reentry clinically HV longer in tachycardia than NSR, but why? According to diagram it COULD theoretically be shorter if no delay in RBB during tachycardia?
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Check this out please #EPeeps @DrFerminGarcia @MelScheinman @DrRoderickTung @MiguelVldrbno
I only seem to be able to find VOM (vein of Marshall) about 50% of time, any suggestions? I am doing occlusive balloon CS venogram with 10 cc full strength dye, possibly too Prox or distal or wrong techniques?@MiguelVldrbno @StevenZweibel
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Thank you both for expert opinions
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Has anyone seen occlusion of vein of Marshall after ablation in CS? Had case where couldn’t find a hint of it after CS RFA. Or, was it just congenitally absent. Interested in thoughts. @MiguelVldrbno @natale_md
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Rotational Flutter around roof scar
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Dot is termination lesion
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Very instructive for fellows. Prior CTI line, flutter looks focal, but fools you because late activation on other side of isthmus “hides” true circuit. Second and third maps after deleting passively activated points.
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Must tap on first photo to see first map
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The only way I can explain this is bundle branch reentry induces from the atrium in a young pt with incredibly enhanced AV nodal conduction, any other ideas?
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