PMC Update (Jun 1, 2026) COVlD levels are "very low" relative to other time periods. 1 in 277 US residents are estimated actively infectious. However, there active hot spots in the Central Appalachia Region, Guam, Franklin County (WA), & Morgan County (AL). 🧵1/5

Data published in npj Dementia shows older Covid survivors experience a 41% increased risk of all-cause dementia and a staggering 77% increased risk of vascular dementia compared to uninfected controls. Leadership and health "care" have abandoned us to this.

Impact of SARS-CoV-2 infection on subclinical myocardial injury in the general population: the Trøndelag Health Study 🚨NORWEGIAN CONFIRMATION BOMBSHELL: COVID infection leaves lasting, hidden scars on the heart muscle, even years later. "An elevated cardiac troponin I (cTnI) level indicates the presence of heart damage!" ➡️Study: - This was a prospective longitudinal cohort study within the Trøndelag Health Study (HUNT), a large population-based survey in Norway, - Researchers measured high-sensitivity cardiac troponin I (hs-cTnI), a sensitive blood marker of subclinical myocardial (heart muscle) injury, at baseline before the COVID-19 pandemic (2017–2019) in 37,823 general-population adults, - The same marker was then re-measured after the pandemic wave (2021–2023) in the 19,550 participants who returned for follow-up, - SARSCoV2 infection status was rigorously determined at follow-up via spike and nucleocapsid IgG antibody tests in blood, combined with self-reported infection history and any available laboratory confirmation of prior infection, - Infection was defined using nucleocapsid IgG (specific to natural infection, not vaccination) plus spike IgG, self-report, and lab confirmation, precisely to capture true infections regardless of vaccination, ➡️Pre-infection result: - Higher baseline hs-cTnI was associated with a lower risk of subsequent SARSCoV2 infection, ➡️Post-infection result: - Confirmed SARSCoV2 infection (any definition) was independently linked to higher post-pandemic hs-cTnI concentrations and a significantly greater probability of an increase in hs-cTnI from pre- to post-pandemic levels, after full adjustment for confounders and baseline troponin, ➡️Vaccination: - Study reports that 98.9% of participants were vaccinated and explain(in Methods) why they used nucleocapsid IgG (not spike) to avoid vaccine confounding, ➡️Limitations: - Correctly sited and commented, - No data on symptoms, asymptomatic/mild/severe cases, or hospitalization, but one may rightfully assume that the majority were mild SarsCoV2 cases, ➡️Conclusion: “SARSCoV2 infection is associated with increased risk of developing chronic subclinical myocardial injury in the general population, but pre-existing chronic subclinical myocardial injury is not associated with increased risk of contracting SARS-CoV-2.” ‼️To all minimiser still shrugging off SARS-CoV-2 as “just a cold” or “over”: this Norwegian study proves every infection silently scars hearts across the general public with lasting subclinical damage, and with the now-established cumulative cardiac injury from reinfections, your denial is quietly killing many! WAKE-UP! #AvoidSars2 #AvoidReinfections academic.oup.com/eurjpc/adva…