世界1・2位をを争う世界有数の科学雑誌PLOS ONEにシャボン玉石けんの論文が載ったそうだ。 なんでも、 石けんの抗ウィルス効果は高く、ヒトインフルエンザにおいては、合成界面活性剤の1000倍の抗ウィルス効果があると。 石けんの威力、すごいんだな〜！ #香害 #柔軟剤危険 #洗剤 #石けん

Studiu PLOS ONE: papagalii captivi par să folosească nume pentru a identifica indivizi - analiză pe peste 880 de înregistrări din proiectul ManyParrots dlvr.it/TT2Q0w #papagali #comunicare #studiu

Show a horse your angry face once. Hours later, after you return with a calm expression, it will still be more guarded around you. Researchers at the Universities of Sussex and Portsmouth published this in Current Biology in 2018. Horses have a memory for emotion. This likely evolved because horses are prey animals. Knowing how the humans around them were feeling, calm or furious, told them whether to stay or run. A 2016 Biology Letters study from Sussex found that horses looking at angry faces show a left-eye gaze bias, where they look at threats through their left eye first. That eye sends signals to the brain's right hemisphere, the one that handles danger. Heart rates go up. This happened just from a flat photograph of a stranger. No movement, no sound, no smell. Just a two-dimensional image of a frown. Sound is tracked separately. A 2018 Hokkaido University study found horses read both faces and voice tone at the same time, the way humans do. Show a horse a happy face paired with an angry voice and it notices the mismatch and stares. They expect what they see and hear to match. Then there is smell. In January 2026, French researchers, publishing in PLOS One, had people watch horror films while wearing cotton pads under their armpits. Those pads, no human present, were placed near horses. The horses startled more easily, kept more distance, showed higher heart rates, all without ever being near the frightened person. Their sweat alone was enough. A horse's heart weighs about 9 pounds. A human heart weighs just over half a pound. After five days of horse therapy, veterans in a Rutgers University study showed measurable reductions in both heart rate and PTSD symptoms. Oxytocin, the bonding hormone, rises in horses when a human stands near them or reaches out to touch them. When that horse pressed against the person in this video, it had already read the face, tracked the tone, smelled the sweat. The warmth was not accidental.

During last decade, articles by Chinese authors contributed a high proportion of academic publications, however, their citation performance remains unclear. The investigation on PLoS journal suggests that those articles' performance is not as good as expected.

Chinese authors have published 863 articles in PLoS Neglected Tropical Diseases, ranking 4th worldwide in terms of publication volume. However, those 863 articles have received an average of 25.88 citations each, ranking 102nd.

Chinese articles published over the last decade exhibit a similar citation performance, ranking about 30th globally, although Chinese authors publish thousands of articles in PLoS ONE every year.

In 2025, PLoS ONE published 19,616 articles, of which 4,735 (about 24%) have a Chinese first author. As of recently, these 4,735 articles have received an average of 1.81 citations each, ranking 34th among the nations that published more than 20 articles in PLoS ONE in 2025.

An investigation reveals that PLoS articles with a Chinese first author have a citation disadvantage. Details: 5gh.org.cn/Report/2026/00001…

Yo siendo Kagome y traigo una bazuca y PLOS naraku. Chao con Adiós

Jubtémnos plos

Sources: 🔹 Geometric Foundations for Collective Quantum Phenomena in Microtubules — Echternach (SSRN, 2026) dx.doi.org/10.2139/ssrn.6502… 🔹 Quantum information flow in microtubule tryptophan networks — Gassab, Pusuluk & Craddock (Entropy, 2026) doi.org/10.3390/e28020204 🔹 Computational predictions of volatile anesthetic interactions with the microtubule cytoskeleton — Craddock et al. (PLOS ONE, 2012) doi.org/10.1371/journal.pone… 🔹 Anesthetic alterations of collective terahertz oscillations in tubulin — Craddock et al. (Scientific Reports, 2017) doi.org/10.1038/s41598-017-0… 🔹 Electronic energy migration in microtubules — Kalra et al. (ACS Central Science, 2023) doi.org/10.1021/acscentsci.2… 🔹 Cytoelectric Coupling — Pinotsis, Fridman & Miller (Progress in Neurobiology, 2023, open access) doi.org/10.1016/j.pneurobio.… 🔹 Microtubules are 'Fractal Time Crystals' — Hameroff, Bandyopadhyay & Lauretta (JCS, 2026) doi.org/10.53765/20512201.33… 17/17

تعرف ليش حساسية الرفض (RSD) عند البالغين أصحاب ADHD تجيك أحياناً كموجة جسدية كاملة، مو إحساس عاطفي بس؟ دراسة كيفية يديدة من Brighton & Sussex Medical School (PLOS ONE ٢٠٢٦) جالست مجموعات تركيزية من بالغين أصحاب ADHD وحلّلت أوصافهم للرفض. النتيجة تقلب الفهم السائد: RSD ليست تجربة عاطفية. إنها تجربة بدنية كاملة. المشاركون وصفوا أعراضاً متطابقة مع استجابة الجسم للقلق الشديد: غثيان، ضغط في الصدر، إحساس بالحرق، أحياناً شلل مؤقت. نفس بصمة الإنذار التي يطلقها الجهاز العصبي عند تهديد حقيقي. والاكتشاف الأقوى: انتظار الرفض أوجع من الرفض نفسه. الدماغ يدفع فاتورة الألم قبل أن يحدث شي. ثلاث ثيمات طلعت من التحليل النوعي: ١) الانسحاب — يتجنّبون تقديم الواجبات، يتراجعون عن التقديم على وظيفة، يقصّرون التواصل مع الأهل والأصحاب. كل ذلك لقطع احتمال الرفض في مهده. الفاتورة المهنية والاجتماعية تتراكم بصمت. ٢) القنّاع — يظهرون "ما يهمّني" بينما الداخل ينهار، فيتعمّق الانفصال العاطفي وفقد الذات. ٣) الإحساس الجسدي — مو ضعف ولا دلال. جهاز عصبي يطلق إنذاراً حقيقياً على تهديد متوقّع، حتى لو ما حصل بعد. د. جيسيكا إيكلس، طبيبة نفسية واستشارية وقائدة الفريق: "المرضى يصفون الألم في أجسامهم، لا في مشاعرهم بس". التطبيق اليومي للبالغ: لما يضيق صدرك قبل اجتماع، أو قبل ما ترسل إيميل، أو قبل ما تقدّم على فرصة، هذا مو جبن. هذا جهاز عصبي يحسبها فاتورة ألم متوقّعة. تسمية الآلية بالاسم تخفّف نصف الحمل، وتفتح باب الفعل رغم الإنذار. doi.org/10.1371/journal.pone… ## #ADHD #Aziz_Al_Khunaizan

鍼灸の抗炎症作用、迷走神経経路から整理する。 Lim et al.（2016, PLOS ONE）の古典的研究では、 ・足三里（ST36）への手鍼刺激がTNF-αの産生を抑制し、この効果が迷走神経切断によって消失することが示された。 ・迷走神経を介したコリン作動性抗炎症経路（CAP）の存在を示す重要。 さらに2026年のFrontiers in Integrative Neuroscienceのレビューでは、電気鍼がlong COVIDに伴う慢性低grade炎症・自律神経失調・免疫代謝異常に対して迷走神経調整を介して作用する可能性が議論されている。 鍼灸師が見るべきポイントとして、 ・足三里への施術は迷走神経→脾臓→マクロファージの経路で全身炎症を制御する ・電気鍼と手鍼では迷走神経への作用が異なる可能性がある ・慢性炎症疾患（膚原病・RA）への理論的根拠としてCAPを活用できる 足三里よく使う先生方、どのような使い方をしますか？

What's your explanation of the prevalence of 'orphan genes'? xunroll.com/thread/148452183… ...doet een poging om zijn geloof staande te houden. Net als 15, 10 en 5 jaar geleden komt hij met het al lang weerlegde GULO gen. Wat is er zo bijzonder aan GULO gen? Het is een gen dat vitamine c helpt aanmaken. Mensen kunnen geen vitamin c meer maken: het gen is stuk. Ook het GULO van de moderne apen is stuk. En die kunnen ook geen vitamine c meer maken (vroeger dus wel). Nu beweren de Darwinisten dat de mutaties in het GULO gen gemeenschappelijke afstamming van alle apen en mensen bewijst. Ik heb dat in 2007 eens nader bekeken. Wat blijkt? Men baseert zich op een heel klein stukje van het GULO gen, namelijk exon X (10). Als we de andere stukjes (exons) van GULO gen bestuderen, dan blijkt dat de afzonderlijke zes exonen van het menselijke GULO pseudogen heel sterk tegen een evolutionaire afstamming pleiten. De 6 andere exons lijken het meest op die van een halfaapje, de lierneus vleermuis, een bladneus vleermuis, een knaagdiertje, de savanneolifant & de witte neushoorn. ... De Darwinisten zijn selectief in hun data analyses. Als je een complete wetenschappelijke analyse uitvoert kom je altijd tot een weerlegging van hun ideologie. Ideologie is de dood voor wetenschap. ... ==================== google translate: xunroll.com/thread/148452183… ...is attempting to maintain his faith. Just like 15, 10, and 5 years ago, he's bringing up the long-debunked GULO gene. What's so special about the GULO gene? It's a gene that helps produce vitamin C. Humans can no longer produce vitamin C: the gene is broken. The GULO of modern apes is also broken. And they can no longer produce vitamin C (they used to). Now the Darwinists claim that the mutations in the GULO gene prove the common descent of all apes and humans. I took a closer look at that in 2007. What do they find? They base their claim on a very small part of the GULO gene, namely exon X (10). If we study the other parts (exons) of the GULO gene, it turns out that the six individual exons of the human GULO pseudogene strongly argue against evolutionary descent. The other six exons most closely resemble those of a prosimian, the lyre-nosed bat, a leaf-nosed bat, a rodent, the African savanna elephant, and the white rhinoceros. ... Darwinists are selective in their data analyses. If you perform a complete scientific analysis, you'll always arrive at a refutation of their ideology. Ideology is the death of science. ... ==================== Peter Borger @BorgerPieter , _Darwin Revisited: Or how to understand biology in the 21st century_ (2018), 243pp., on 182, 213 amazon.com/Darwin-Revisited-… ==================== Het GULO pseudogen van René en Gerdien: vooringenomenheid en dataselectie door Peter Borger | 26 november 2019 logos.nl/het-gulo-pseudogen-… google translate: ... So I'll keep it short. In my book "Back to the Origins" (2009), I devoted a short chapter to the philosophy of science (chapter 2). On page 24, I wrote: "It's not difficult to find evidence in favor of a theory. If one only seeks evidence that supports a particular hypothesis, denying or rejecting all disconfirming observations, then it's possible to defend even the most ridiculous hypotheses. This is called proof by verification. By simply verifying a proposition, it's possible to argue that the Earth is flat, that the Earth is young, that it's old, that it's green, or that it's blue. It doesn't matter what hypothesis you take, you can always find a verifying example that 'proves' it! Evidence obtained by verification doesn't really support a scientific theory." So, it's not important that our article supports (or proves) the aforementioned hypotheses. What matters is that there's a tremendous amount of evidence against these hypotheses. The point is that such negative evidence carries much more weight, because negative evidence refutes hypotheses. In my book and articles, I've highlighted this negative evidence in more detail, so that people hear a different perspective. The public is only shown a certain portion of the data, a selection that creates the impression that common descent is a fact. My book shattered that illusion and explained biology in an alternative, non-Darwinian way. Similarly, Royal Truman and I explained the GULO pseudogene in an alternative, non-Darwinian way. Shouldn't there be alternative explanations for biological phenomena? Is that what science dictates? No, it has nothing to do with science. It does have to do with the Darwinist's bias. The Darwinist apparently knows everything beforehand. But why should we even do research if we already know everything beforehand? Suppose the new biological data refutes Darwin's universal common descent, what then? That's impossible, says the Darwinist, because they already know beforehand that it's impossible. Is that a scientific attitude? No, it's bias, which kills the urge for further, in-depth research in the bud. This bias is anti-scientific and has already led to major delays in science.3 Biology as a whole refutes universal common descent Royal Truman and I saw in 2006 that universal common descent is absolutely untenable. If you view an organism as a holistic information system (and not as a collection of mutations in individual selectable genes), then common descent is impossible. This is actually quite easy to understand. We have discovered that the genomes of all species are characterized by genetic information found nowhere else: orphan genes. Humans and chimpanzees differ by 1,414 protein-coding genes.4 and approximately the same number of microRNA-coding genes5, of which 130 families were confirmed as unique human information.6 A comparison of plant DNA reveals 1179 gene families, which are found only in angiosperms.7 Scientists discovered more than 28 thousand genes that are unique to ants and not found in other insects.8 The squid genomes are characterized by hundreds of unique genes.9 And so on, and so on. In all genomes analyzed to date, "orphan genes" account for approximately 10-30% of the identified genes. The sudden appearance of new genetic information, its functional complexity, and its integration into the genome have no plausible Darwinian explanation. This unique information, devoid of developmental history, precludes a stepwise, selection-driven evolutionary process. If we take these 10-30% unique genes seriously, as Royal Truman and I do, then there can be no question of universal common descent. And then humans and chimps cannot share an ancestor, because they are characterized by a yawning information gap that cannot be filled with mutation-selection narratives. The only thing left for us is to find an alternative explanation for data that does suggest universal common descent. And that is what Royal Truman and I did for the GULO pseudogene. Is that permissible? Yes, it is, because science is thankfully still a free enterprise (although it is increasingly becoming a politically correct Darwinian system). The functioning GULO gene, as found in the rat, consists of twelve exons. Exons are the coding sequences that must be joined together to produce the GULO protein. In humans, only six of these exons are found. For the genetic analyses, only Exon X (one of the six exons found in humans) was compared and this appears to indicate a line of descent, as expected based on Darwinian considerations. A more in-depth genetic analysis of the individual exons that make up the GULO gene was published in 2014. The six individual exons of the human GULO pseudogene argue strongly against an evolutionary descent. They most resemble those of a prosimian, the lyre-nosed bat, a leaf-nosed bat, a rodent, the African bush elephant, and the white rhinoceros.10 Renè Fransen and Gerdien de Jong therefore rely on outdated and selective information. The fact that humans and apes have the exact same inactivating mutation in the GULO gene can't be a coincidence, of course. And it isn't. Thanks to the large-scale genome projects of the past decade, we now know that a large proportion of mutations occur in hotspots-- an observation no one expected.11 In the genomes of humans and rhesus monkeys, mutations were even observed to accumulate independently at exactly the same sites, thus creating an illusion of common descent. The alternative explanation for the GULO pseudogene, which we proposed in 2007, therefore appears to be correct. At the very least, it is a scientific alternative. In light of the thousands of orphan genes we find in humans and animals, it is even the best explanation. It was to be expected that this would not be to everyone's liking. Footnotes 1 Another strong piece of evidence has always been chromosome 2, which appears to be a fusion of two chromosomes, which occur separately in apes. But this evidence has been refuted by the latest biological data, which show that a functional gene resides precisely at the site of the hypothetical fusion. 2 sterrenstof.info/het-vitamin… . 3 Consider, for example, Mendel or McClintock, whose biological insights were blocked for many decades by the Darwinians. Or consider Darwinian "junk DNA," which has proven to be a huge science-stopper. 4 Ruiz-Orera J, Hernandez-Rodriguez J, Chiva C, et al. Origins of De Novo Genes in Human and Chimpanzee. PLoS Genet. 2015;11(12):e1005721. Published 2015 Dec 31. doi:10.1371/journal.pgen.1005721 5 mirnablog.com/how-many-uniqu… . 6 personal communication with Bastian Fromm, University of Stockholm. 7 Science. 2013 Dec 20;342(6165):1241089. doi: 10.1126/science.1241089. The Amborella genome and the evolution of flowering plants. Amborella Genome Project. 8 Genome Res. 2013 Aug;23(8):1235-47. doi: 10.1101/gr.155408.113. Epub 2013 May 1. Social insect genomes exhibit dramatic evolution in gene composition and regulation while preserving regulatory features linked to sociality. Simola DF1, Wissler L, Donahue G, Waterhouse RM, Helmkampf M, Roux J, Nygaard S, Glastad KM, Hagen DE, Viljakainen L, Reese JT, Hunt BG, Graur D, Elhaik E, Kriventseva EV, Wen J, Parker BJ, Cash E, Privman E, Childers CP, Muñoz-Torres MC, Boomsma JJ, Bornberg-Bauer E, Currie CR, Elsik CG, Suen G, Goodisman MA, Keller L, Liebig J, Rawls A, Reinberg D, Smith CD, Smith CR, Tsutsui N, Wurm Y, Zdobnov EM, Berger SL, Gadau J. 9 Nature. 2015 Aug 13;524(7564):220-4. doi:10.1038/nature14668. The octopus genome and the evolution of cephalopod neural and morphological novelties. Albertin CB1, Simakov O2, Mitros T3, Wang ZY4, Pungor JR4, Edsinger-Gonzales E5, Brenner S6, Ragsdale CW7, Rokhsar DS8. 10 Tomkins JP. The Human GULO Pseudogene-- Evidence for Evolutionary Discontinuity and Genetic Entropy. Answers Research Journal 7 (2014):91–101. answersingenesis.org/content… 11 exac.broadinstitute.org. 12 Rhesus Macaque Genome Sequencing and Analysis Consortium, Evolutionary and Biomedical Insights from the Rhesus Macaque Genome, Science 316:222–234, 2007.