Citations
-Lee C, et al. (2015). The mitochondrial-derived peptide MOTS-c promotes metabolic homeostasis and reduces obesity and insulin resistance. Cell Metabolism, 21(3), 443–454.
-Reynolds JC, et al. (2021). MOTS-c is an exercise-induced mitochondrial-encoded regulator of age-dependent physical decline and muscle homeostasis. Nature Communications, 12, 470.
-Wang M, et al. (2023). MOTS-c repairs myocardial damage by inhibiting the CCN1/ERK1/2/EGR1 pathway in diabetic rats. Frontiers in Nutrition, 10, 1060684.
-Domin R, et al. (2023). MOTS-c serum concentration positively correlates with lower-body muscle strength and is not related to maximal oxygen uptake — a preliminary study. International Journal of Molecular Sciences, 24(19), 14951.
-Lu H, et al. (2024). The mitochondrial genome-encoded peptide MOTS-c alleviates nonalcoholic steatohepatitis by inhibiting inflammation and apoptosis. Cell Reports (related NASH studies).
-Zheng Y, et al. (2023). MOTS-c: A promising mitochondrial-derived peptide for metabolic disorders and aging. Frontiers in Endocrinology (mechanistic review).
-Wan W, et al. (2023). MOTS-c: Effects and mechanisms related to stress, metabolism and aging. Journal of Translational Medicine, 21, 885.