What woke fnut booked it in the first place??? It's an elementary school FFS.

Well if you started mass deportations and looked after us. Then people wouldn’t have to act this way. Your meant to protect US BRITISH NOT THE FOREIGN CRIMINALS. Also sort out our defence you idiot. We could get attacked and because your a fnut we could lose. Resign

🆕 Plant-based caloric restriction diets versus conventional calorie-restricted diets for weight loss and metabolic health in obese adults: a 12-week randomized, open-label, non-inferiority trial. Front. Nutr. 13:1805225. doi: 10.3389/fnut.2026.1805225 doi.org/10.3389/fnut.2026.18…

Wow. Who saw this coming. Starts with a kiss and then we all know where it goes. Whoever hired this fnut needs to be canned.

Jones 2025 A Generic Model & Conc Framework to Prime Curiosity Across Health & Soc Care.. J. Eval Clin Prac. doi.org/10.1111/jep.70252 Wirnitzer et al Toward a roadmap for addressing today's health dilemma.. Front in Nutr DOI:10.3389/fnut.2025.1676080 2026 doi.org/10.1111/jphd.70034

Imagine immigrating to America and then having the arrogance to then threaten good Americans?! 🇺🇸 No, newbie fnut, we will be coming for you, hating ingrate!

No one will go down worse than idiot child low IQ fnut Justin Trudeau. Just a complete moronic woke turd. Mark Carney might be follow up.

Not all magnesium is the same. A new RCT found magnesium L-threonate was linked to improved cognition, working memory, reaction time, HRV, and estimated cognitive brain age after 6 weeks. That’s why I always say: form and quality matter. Study: 10.3389/fnut.2025.1729164

Thank you for all your doubts and criticisms. I have added the latest eighth piece of evidence: 8. Tests on C57BL/6N mice susceptible to diet-induced obesity, type 2 diabetes and atherosclerosis showed that lifelong supplementation of sucrose accounting for 10-25% of total calories in staple foods did not cause overweight and obesity in male C57BL/6N mice, with normal blood glucose, plasma total cholesterol and hepatic triglyceride levels [doi: 10.3389/fnut.2022.1076073]. Accordingly, sugar alone does not raise the risk of type 2 diabetes. In reality, however, people rarely consume pure sugar or add sugar to staple foods. Sugar is mostly incorporated into beverages, pastries, baked biscuits and other foods. The appealing taste of desserts drives increased intake of sugary products, which leads to excessive calorie intake as well as overconsumption of other ingredients. For instance, baked goods contain acrylamide. Animal studies have proven that long-term acrylamide intake triggers pancreatic islet cell apoptosis, elevates fasting blood glucose, and induces oxidative stress and inflammatory responses, all of which are pivotal drivers of the onset and progression of type 2 diabetes.

Accumulation of glycated proteins is undoubtedly a consequence rather than a driving cause of aging. This view is supported by multiple research findings. Nematodes survive 20 days on a regular diet, while their lifespan extends to 24 days under high-glucose feeding (doi:10.1038/s41467-022-33630-0). In mouse experiments, the first death occurred at 78 weeks in the group with sugar accounting for 10% of calorie intake, compared with 87 weeks in the 25% sugar calorie group (doi: 10.3389/fnut.2022.1076073). For humans, moderate dietary sugar intake cuts the early death risk by approximately 10% to 20% (doi: 10.1093/ajcn/nqy268). Higher consumption of whole fruits such as blueberries, grapes and apples is linked to reduced risk of type 2 diabetes, whereas increased intake of fruit juice correlates with higher disease risk (doi: 10.1136/bmj.f5001). A large European prospective cohort study further revealed that higher dietary intake of MGO and 3-DG, precursors of advanced glycation end products (AGEs), does not elevate type 2 diabetes risk, but is associated with lower incidence of the disease (doi: 10.1007/s00394-026-03904-0). Collectively, these evidence proves that glycated protein buildup is merely a result of aging, not its inducing factor.