OBAMA was involved in the CREATION of U.S. BIOLABS conducting gain-of-function research of deadly pathogens across the world, especially in Ukraine using U.S. taxpayer dollars, through agencies like USAID. After the bioweapons convention ban ended in 2001 and after the anthrax attacks, Dr. Fauci essentially restarted the bioweapons arms race under the pretension of developing vaccines. In 2014, Obama moved Fauci's gain-of-function research offshore after Obama was essentially begged to shut down operations from over 250 scientists because of the dangerous research Dr. Fauci was conducting. President Trump was the one who shut down gain-of-function research in the U.S. and abroad. DNI Tulsi Gabbard first exposed these labs in 2022 and now the proof of 120 biolabs in 30 countries, with a heavy focus on Ukraine, where the U.S. built and supported over 40 labs has been released and it all goes right back to the Obama administration and Dr. Fauci.

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CONFIRMED: Barack Obama Was Involved in Creation of US Biolabs Handling Especially Dangerous Pathogens in Ukraine. Obama truly is an evil person. There are at least ten documents implicating Obamas Department of Defense in funding the Ukrainian Biolabs. The US Embassy even scrubbed documents from their website that shows the US Department of Defense funded the creation of a Ukrainian diagnostic biolab. They were found in archives. thegatewaypundit.com/2026/06…

40 US-FUNDED LABS IN UKRAINE STOCKED WITH DEADLIEST PATHOGENS Declassified documents confirm over 40 US-funded biolabs across Ukraine stored bioweapons including: - Anthrax - Tularemia - Tuberculosis - African Swine Fever - Newcastle Disease - MERS & SARS - Marburg - Ebola These labs were part of networks directly tied to US entities: - Black & Veatch - CDC - Metabiota - EcoHealth Alliance - Battelle - CH2M Hill - Ukrainian Ministry of Health US DoD contractors Maps in the docs show the sprawling network. This wasn’t “health research” — this was a massive biolab empire on Russia’s border, funded by American taxpayers. Who authorized this? Why were these deadly pathogens there? And why did the media and officials deny it for years while calling it “Russian disinformation”? This needs full Congressional investigation NOW. Heads must roll. America deserves answers. The world deserves the truth.

‼️🚨Russian Defense Ministry is investigating 240 disease causing pathogens discovered in US-sponsored biolabs in Ukraine including anthrax and cholera amongst others… Pathogens are studied and developed with intent of use for "offensive actions."

The -not specific to Ukraine- admission of gain of function research is new though, isn't it? The US unit that organized the so called dismantling of USSR-era pathogens denied any GoF activities previously.

🔴I agree with the general idea that elevated IgG titers to latent or persistent pathogens should not simply be dismissed as meaningless. But I would add a few mechanistic clarifications. First, not all herpesviruses establish latency in B lymphocytes as their main reservoir. EBV is the classic herpesvirus with a major B-cell reservoir. But CMV, HHV-6, VZV and other herpesviruses have different latency biology and different cellular or tissue reservoirs. So it is important not to generalize EBV biology to all herpesviruses. Second, latency is not simply a state in which infected cells become completely invisible or biologically inactive. This is especially clear with EBV. EBV has different latency programs. Latency 0 is the most immunologically silent state, but latency I, II and III still express viral antigens to different degrees, and these infected cells can be controlled by EBV-specific CD4 and CD8 T cells. For example, latency I, classically seen in Burkitt lymphoma, expresses EBNA1, and EBNA1-specific CD4 T-cell responses are part of the immune control of EBV-infected cells. But the same general principle applies more broadly: many latent or persistent pathogens are not simply “doing nothing.” Latently infected or persistently infected cells can express viral non-coding RNAs, microRNAs, latency-associated transcripts or other regulatory RNA species that can modulate immune sensing, inflammation, cell survival, antiviral responses and immune evasion. In herpesviruses, viral microRNAs and non-coding RNAs can help maintain latency, suppress lytic genes, reduce immune recognition, alter host-cell survival and shape the local inflammatory environment. Even latently infected EBV cells can express non-coding viral RNAs such as EBERs and EBV microRNAs. These can modulate inflammation, interfere with antiviral sensing, shape B-cell survival and contribute to immune evasion. EBV also has immunomodulatory strategies, including viral IL-10-like activity mainly associated with lytic or reactivation contexts, which can help dampen antiviral immune responses and reduce immune recognition. So “latent” does not necessarily mean silent, harmless or irrelevant. It often means that the virus is being actively contained by immune surveillance. In healthy people, the key point is not that latent viruses become totally invisible. The key point is that CD4 and CD8 T-cell surveillance, NK-cell function and other antiviral mechanisms keep these reservoirs highly contained. This is why most healthy carriers do not have constant clinically relevant reactivation. The pathogen is usually controlled, restricted to limited reservoirs, and only reactivates more meaningfully during periods of immune stress, acute infection, immunosuppression or loss of immune control. Where things become more interesting is in genetically susceptible individuals. Immunogenetic and evolutionary studies suggest that some ancestral HLA-II haplotypes, especially DR2-DQ6, DR3-DQ2 and DR4-DQ8, have broader or more promiscuous peptide-binding repertoires than many other HLA-II backgrounds. This broader antigen-presentation capacity may have been advantageous during acute infections, because it allowed stronger and more diverse CD4 T-cell responses against pathogens. But if the pathogen persists, reactivates, or uses latency/evasion mechanisms, the immune system can remain chronically stimulated. That chronic stimulation can induce sustained IFN-γ, tissue inflammation, ectopic HLA-II expression in tissues that do not normally present antigen at high levels, and continuous presentation of both pathogen-derived and self-derived peptides. In that environment, the probability of activating autoreactive T and B cells increases. This is where HLA background matters. 1/3 Continued in the next post.👇🏻

Y'all are such idiots. A biolab does not equal biological weapons. Yes, dangerous pathogens are kept in some and those need to be kept in secret locations. How do you think antibiotics or vaccines are developed? On a chalkboard???