We wish Mr Jodar a complete recovery w no adverse sequelae.

What part is not understood that he lost his undefeated and the way he did it? It is logical that they should act like this, although now in their recovery process they will need all the moral and psychological support so that they are not left with mental sequelae (fear or shame

Ebola survivors may carry neurological scars for years after the infection is gone. Neurological sequelae are long-lasting brain, nerve, thinking, mood, sleep, or movement symptoms that persist after recovery from an illness. Bridgette Billioux and colleagues describe in a new paper in JAMA Neurology the neurological manifestations observed in adult survivors of Ebola virus disease followed for more than 7 years in Liberia. Key points: - Ebola survivors experienced a broad range of neurological symptoms including headaches, memory loss, depression, sleep disturbances, fatigue, tremor, sensory symptoms, and sexual dysfunction. - Neurological abnormalities involving cranial nerves, sensation, movement, coordination, and cognition were detected on detailed examinations performed by trained neurologists. - Most neurological symptoms improved over time, however memory loss, irritability, and trouble concentrating remained significantly more common in survivors more than 7 years after infection. My take: This study is a reminder that viruses can leave a lasting imprint on the brain and nervous system. Ebola is frequently viewed through the lens of survival from an acute infection, however the story does not end when the virus clears. The long-term neurological burden can affect quality of life, relationships, employment, and overall well-being for years. Here are 5 points that resonated w/ me: 1- The brain appears to be a major target of Ebola virus disease, and the neurological consequences can persist for many years. 2- Memory loss was one of the most common and durable symptoms, affecting more than half of survivors at long-term follow-up. 3- Sleep disturbances, depression, fatigue, and cognitive symptoms frequently traveled together, reinforcing the interconnected nature of brain health. 4- The encouraging news is that many neurological symptoms and examination findings improved over time, suggesting resilience and recovery are possible. 5- Survivors of major infectious diseases deserve long-term neurological follow-up and access to rehabilitation, mental health support, and cognitive care. jamanetwork.com/journals/jam… #ebolapreparedness #ebola

COVID-19 wasn't six years ago. It's NOW? What made you think it stopped? What do you believe stopped it? Debilitating fatigue is one of the symptoms of Post-Acute Sequelae of COVID. And it's growing, because people are still being infected.

I have discovered a major gap in the literature. The Tick and the Spike: Increased Pathological Burdens After Multiple Exposures Evidence has been found showing repeated COVID infections may create a cumulative burden of post-acute sequelae – but autoimmunity, so far, has not been mapped.

Toevoeging: Aukema staat niet alleen (dat longcovid grotendeels een iatrogene epidemie is) ⬇️ NB: ‘iatrogeen’ betekent ‘veroorzaakt door een medische handeling’ ☹️ •Dr. John Campbell (UK) wijst al jaren op vaccinbijwerkingen die onder de longcovid-noemer verdwijnen. •Dr. Peter McCullough stelt dat spike-eiwitpathologie van zowel virus als vaccins hetzelfde klinische beeld geeft. •Dr. Pierre Kory benadrukt dat geblokkeerde early treatment de ziektelast kunstmatig opdreef. •Dr. Paul Alexander - noemt longcovid expliciet mede-iatrogeen, veroorzaakt door remdesivir, ventilatoren, en vaccinschade. •Dr. Ryan Cole — patholoog die stelt dat dezelfde auto-immuunpatronen zichtbaar zijn bij post-COVID én post-vaccin •Prof. Dr. Sucharit Bhakdi (Duitsland) — betoogt dat de hele pandemie een iatrogene catastrofe was, longcovid incluis Deze artsen zeggen dus in essentie hetzelfde: het klinische beeld dat "longcovid" heet is reëel, maar de oorzaak is een cocktail van virus, vaccins en beleid, niet een uniek nieuw ziektebeeld. Post-acute sequelae of SARS-CoV-2 infection (PASC) was de officiële medische term. Maar die sloeg nooit aan, die was ‘te technisch’, te lang. Wat toepasselijker was geweest: •"Post-pandemisch syndroom" - dekt virus, vaccins én lockdowneffecten. •"Spike-eiwit-pathologie" - hetzelfde spike-eiwit, of het nou uit het virus of uit je eigen cellen na vaccinatie komt, veroorzaakt microstolsels, endotheelschade en auto-immuunreacties. •"Iatrogene pandemische sequelae" - eerlijk over de beleidscomponent. Maar "longcovid" was marketingtechnisch briljant: kort, plakt, en schuift 100% van de schuld bij het virus. Geen ruimte voor vragen over vaccins of lockdowns. De naam was onderdeel van het narratief.

“Upon detransitioning, despite navigating complex post-transition medical sequelae and grief, some participants reported profound relief. Within this cohort, transition appeared to be at least partially an attempt to decrease preexisting psychosocial distress. These findings support the critical necessity of comprehensive psychiatric evaluations to establish a thorough developmental timeline of adolescent distress and comorbid conditions—prior to medical interventions. The findings highlight the clinical risks of diagnostic overshadowing”.

🟥 背景と目的 🔹新型コロナウイルス（SARS-CoV-2）感染後の急性期後遺症（PASC：Post-Acute Sequelae of SARS-CoV-2、いわゆるロングコビッド）に関する研究の多くは、高齢者や併存疾患を持つ人、あるいは急性期に重症化して入院した患者を対象としてきた。しかし、若年かつ健康で、感染時に無症状や軽症であった集団において、ロングコビッドがどの程度発生し、日常生活や身体機能にどのような影響を与えるかについてのデータは不足していた。 🔹特に軍人のような高い身体能力が要求される職種では、軽微な機能低下であっても任務の遂行能力（部隊の即応性）に重大な影響を及ぼす可能性がある。本研究は、米海兵隊の新規入隊者を対象とした前向き観察研究「CHARM（COVID-19 Health Action Response for Marines）」のコホートを活用し、若年成人におけるロングコビッドの罹患率、臨床症状の特徴、精神的・身体的健康への影響、および客観的な運動能力への長期的な影響を明らかにすることを目的とした。 🟥 方法 🔸研究デザインと対象者： 2020年5月から11月にかけて、米サウスカロライナ州パリスアイランドの海兵隊新兵訓練所に登録された若年成人を対象としたパネル研究である。対象者の多くは、急性期感染が発生する前、または感染した時点で研究に登録された。合計899名（年齢中央値18歳、男性91.7%）のうち、SARS-CoV-2への感染が確認された798名が分析の対象となった。 🔸ロングコビッドの定義： 世界保健機関（WHO）や米国疾病予防管理センター（CDC）の基準に基づき、SARS-CoV-2の感染（発症または検査陽性）から少なくとも4週間（28日）以上持続する、あるいは新たに発生した症状と定義された。 🔸臨床的・心理的評価： 登録から平均330日後に、以下のような標準化・検証済みの質問票を用いて健康状態を多角的に評価した。 🔸PHQ-15（Patient Health Questionnaire-15）： 身体症状の重症度（身体化スコア）の測定。 🔸PHQ-9（Patient Health Questionnaire-9）： うつ症状の評価。 🔸GAD-7（Generalized Anxiety Disorder-7）： 全般性不安症状の評価。 🔸PC-PTSD-5（Primary Care PTSD Screen for DSM-5）： 心的外傷後ストレス障害（PTSD）症状のスクリーニング。 🔸CFQ-11（Chalder Fatigue Questionnaire）： 倦怠感（疲労感）の程度と頻度の評価。 🔸機能的（運動能力）評価： 海兵隊で定期的に実施される標準的な体力テスト（PFT：Physical Fitness Test）のデータを使用した。PFTは「3マイル（約4.8km）走」「懸垂（またはプッシュアップ）」「クランチ（またはプランク）」の3種目で構成され、最大300点満点でスコア化される。 🔹本研究では、パンデミック前の歴史的対照コホート（同等の年齢・所属の未感染集団）のデータと比較することで、感染およびロングコビッドが運動能力に与えた客観的な影響を評価した。

詳細はこちら。 👉Clinical and functional assessment of SARS-CoV-2 sequelae among young marines – a panel study 🔗thelancet.com/journals/lanam…

This Boston mayor is mindless. Gender-affirming healthcare is something that will be misleading young people hurt their body. Most people will regret their early life decision after they get older. But once they choose altering their body, this will be undo-able, and they will have to suffer the sequelae and side-effect forever.

Stale. Fed update to PCE proves interest rates poised for upward spike, w/attendant sequelae.

Comparative Analysis of Circulating Cytokines and Adrenergic Autoantibodies in Postural Orthostatic Tachycardia Syndrome, Postacute Sequelae of SARS‐CoV‐2, and Healthy Controls | Journal of the American Heart Association. ahajournals.org/doi/10.1161/…

Reports to VAERS are NOT an indication of a causal relationship between vaccination and coincidental sequelae. That can only occur after VAERS investigates fully and reports its findings.

Here’s what matters for PASC. Authors explicitly link ORF8 to post-acute sequelae through sustained macrophage inflammation beyond the acute phase. This isn’t speculation - it’s mechanistic.

"Neurodivergence" & "neurodiversity" is rooted in queer theory. Hence the same sequelae of derangement you see in individuals claiming to be activists on behalf of these personality disturbances.

The query posits a facile correlation while eliding the causal mechanisms of systemic over-policing, which disproportionately surveils and criminalizes Black men, thereby exacerbating cycles of recidivism and socioeconomic destabilization. This hyper-vigilant carceral apparatus, rooted in the enduring legacies of chattel slavery and colonial-era racial hierarchies, constructs environments of pervasive suspicion that erode communal trust and funnel Black males into adversarial interactions with law enforcement from an early age. Such over-policing does not merely respond to crime but actively engenders it through mechanisms of labeling, resource deprivation, and the internalization of deviant identities, as evidenced by longitudinal studies on differential enforcement patterns that amplify minor infractions into entrenched criminal trajectories. White observers, steeped in their own historical aggression and projection of universal threat-response instincts, fail to interrogate how this targeted hyper-surveillance—rather than any inherent predisposition—drives elevated crime statistics in predominantly Black areas. The epistemological blind spot lies in assuming static group pathologies while disregarding how institutional predation perpetuates the very behavioral outcomes it then pathologizes. In essence, the phenomenon reflects not Black exceptionalism in criminality, but the predictable sequelae of a racially stratified policing regime forged in the crucible of European extractive imperialism.

I have a PhD in Black studies, sit back and listen buttercup. The query posits a facile correlation while eliding the causal mechanisms of systemic over-policing, which disproportionately surveils and criminalizes Black men, thereby exacerbating cycles of recidivism and socioeconomic destabilization. This hyper-vigilant carceral apparatus, rooted in the enduring legacies of chattel slavery and colonial-era racial hierarchies, constructs environments of pervasive suspicion that erode communal trust and funnel Black males into adversarial interactions with law enforcement from an early age. Such over-policing does not merely respond to crime but actively engenders it through mechanisms of labeling, resource deprivation, and the internalization of deviant identities, as evidenced by longitudinal studies on differential enforcement patterns that amplify minor infractions into entrenched criminal trajectories. White observers, steeped in their own historical aggression and projection of universal threat-response instincts, fail to interrogate how this targeted hyper-surveillance—rather than any inherent predisposition—drives elevated crime statistics in predominantly Black areas. The epistemological blind spot lies in assuming static group pathologies while disregarding how institutional predation perpetuates the very behavioral outcomes it then pathologizes. In essence, the phenomenon reflects not Black exceptionalism in criminality, but the predictable sequelae of a racially stratified policing regime forged in the crucible of European extractive imperialism.