Y Not? retweeted
Red light green tea powerfully reduces skin aging in clinical trial.
This is one month of progress utilizing:
◇ Green tea filled cotton pads applied to the face
◇ 670 nm red light
For 20 mins per day.
Both tools work on the key paths found in skin aging - collagen/elastin preservation synthesis, reduction of ROS.
26 Aug 2025
Red light therapy reverses all signs of skin aging, as shown in a recent study.
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Uma Mahesh(Ntr Bhaktudu) retweeted
ASVR is aging Like fine wine
Tigeruuuu choose ur Director ,fitness mentor wisely
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nayaa retweeted
I think it makes sense. He's aging out of modeling, has to do odd jobs for money, barely has any family
Jun 12
i’ve seen so many astrologers/ tarot readers saying that bryce is very sad and losing hope in life 😭
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Ppl forget Beyonce got a lil touch of white in here and it's starting to show aging like cheese
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Eda kelava theres something called as time progression and aging. You cant just prop up young raw talents at this median age without institutional support. Monnakalodu tharkkikkan poya enikk venam 😭😭😭
Morons have no brains. So they blabber to fill the emptiness in the head.
They say union government doesn’t support Kerala.
Morons won’t say “union” didn’t support Kerala back in 1980s, too.
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k #FreePalestine🍉 retweeted
that’s usually how aging looks
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Certified Boogeyman
MCoBeauty – Miracle Anti-Aging Repair Serum – Nighttime Renewal Formula with Hyaluronic Acid, Arginine & Antioxidants for Radiant, Youthful Skin – Face Skin Care
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Shinya Goto retweeted
Jun 12
Loss of STMP1 perturbs mitochondrial cristae and drives cellular inflammation, heart failure and cardiac aging. ahajrnls.org/4aKmdfw
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Alison Jacobson retweeted
Sinclair's new preprint provides the first direct experimental evidence for the Information Theory of Aging: your cells don't age because they break — they age because they forget who they are. The biological blueprint is still intact. The cells just stop reading it correctly. That distinction changes the entire target: you don't repair damage. You restore data. And data can be restored.
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FAILED MARSHAL (Resurrected) retweeted
Sounds like excuses for not replacing the aging fleet.
#WATCH | Greater Noida, UP | On IAF's AN-32 aircraft accident in Assam, Air Marshal Sanjeev Kapoor (Retd) says, "The exact reason will be known as and when more details come out or the Court of Inquirydetails are made public."
"AN-32 is a very safe aeroplane which has been flying in the Indian Air Force for last 40 years. It has been the backbone of the transport fleet operating in the north in Siachen, Leh, Ladakh and drop zones. Operating over the oceans in Andaman and Nicobar and sustaining our island territories of Lakshadweep and Port Blair. It has also been operating in the desert areas in the Southwestern Air Command. It has been operating in the maritime roles, along our coastal regions. So long and short, be it para, be it assault, be it any evacuation, special ops, or a normal communication, these aeroplanes have stood the test of time for the Indian Air Force. They have been upgraded. They are absolutely in good condition, though old."
Five Indian Air Force personnel have lost their lives in the air crash involving the AN-32 transport aircraft at the Jorhat air base in Assam.
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YousAChopHeaux retweeted
She’s definitely aging the best out of her peers…..
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Rayan DylanH retweeted
Jun 12
NEW PREPRINT: Scientists may have found direct evidence that aging is driven by the loss of cellular information, not just the accumulation of damage
For decades we've focused on what aging cells accumulate. This paper focuses on what they lose: Information.
Using a new technology called SeqTag, researchers measured gene expression, chromatin accessibility, and histone modifications in the same aging cells.
What they found was striking: the regulatory systems that tell cells who they are become increasingly out of sync with age. The authors call this "molecular asynchrony."
As cells age, chromatin structure, histone marks, and gene expression begin drifting apart. Regulatory entropy rises. Repressive chromatin erodes. Cells become less certain of their identity and more likely to drift toward alternative fates.
This is what the Information Theory of Aging (ITOA) states: that aging occurs when cells lose epigenetic information, the instructions that tell the genome how to maintain youthful function. DNA may remain largely unchanged, but the system that reads it gradually loses fidelity.
What's remarkable is that this paper doesn't just describe this phenomenon. It quantifies it. The authors measure increasing regulatory entropy, loss of H3K27me3-mediated repression, erosion of heterochromatin, weakening lineage fidelity, and increased cell-fate drift during aging. In progenitor cells, the barriers that normally preserve cellular identity become progressively weaker with age.
Mechanistically, the study argues that aging is associated with increasing molecular asynchrony between chromatin accessibility, H3K27ac/H3K27me3 remodeling, and transcriptional state.
This decoupling is accompanied by increased regulatory entropy, loss of repressive chromatin architecture, and weakening of lineage constraints. Genes affected are those involved in chromatin organization, DNA damage, and Wnt signaling, consistent with ITOA.
Importantly, the authors provide quantitative evidence that age-related heterochromatin erosion lowers the energetic barriers that maintain cell identity, offering a potential mechanistic link between epigenetic information loss, cell-fate drift, and late-life disease susceptibility 👏
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There’s a moment when the noise drops away and one clear line rises a bit higher, a tad cleaner, until the only thing left is pure ascent. That’s the feeling we’re engineering at Algomash.
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